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Z., Li, C., Wang, S., Xiong, T., Nie, S

430 These findings suggest that iron metabolism is abnormal in the cells of Friedreichs Ataxia models, and regulation of iron metabolism may have some ameliorative effects on Friedreichs Ataxia

Naik, et al

eNAP-2 was related to the rat WDNM1 protein which is included in a four-disulfide core family of proteins with diverse functions and a homology to human antileukoproteases [32]